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Proof About Cholesterol
By EricHanson | November 19, 2009
In the Spring of 1958,a team of Harvard medical scientists furnished the final, incontrovertible proof that a surplus of cholesterol in the blood stream can, and does, cause coronary disease. Although this is quite a long time ago, the facts remain the same.
The scientists took little bits of blood vessel wall tissue, and kept them alive in a culture medium (which, in a real sense, means an artificial blood). They then began to add tiny drops of cholesterol to the feeding mixture—which, let us remind you again, corresponds to blood. In four or five days, fat deposits formed in the blood vessel cells.
By check-testing, they soon found that the size of the fat deposit in the blood vessel wall was directly proportional to the amount of cholesterol added. If the blood vessel cells were allowed to remain in the cholesterol-fed medium, the fat deposits soon increased to such size that the blood vessel cells finally withered and died.
Mind you, the doctors actually watched cholesterol produce blood vessel disease, but the experiments went much further. They found that satt-rich stearic acid, which occurs in certain animal fats, aided and abetted the formation of fatty deposits and caused them to increase rapidly and markedly in size.
Then the researchers did an even more astonishing thing. They added linolenic acid, a rich, primary source of polys, to the culture medium, and cholesterol as before; but the fat deposits didn’t form! Thus, they proved experimentally, with live human tissue, that the linolenic-acid polys could offset and counteract the effects of cholesterol, and keep it from forming deposits in the blood vessel walls.
The team continued the experiment. Was the action of the linolenic-acid polys more than protective and preventive? Would the polys actually decrease the size of the deposits which had been laid down in blood vessel cells? This is what they found.
Once a deposit of fat had been formed, the more linolenic acid added to the culture medium, the more the size of the deposit decreased. This can also be interpreted in another sense; viz., the more polys in a fat, the better it will work in preventing “The Grease Trap Disease,” and diminishing the size of fat deposits already formed.
However, don’t take this to mean that you can simply swallow linolenic acid, or any other rich source of polys, and magically cure yourself of coronary disease.
Your fat intake still has to be “balanced.” Remember that an excess of any kind of fat droplets in the blood stream can also cause trouble; and remember, too, that the blood becomes sticky and sludgy, and clots more readily, when you eat too much of any kind of fat. So, keep your total fat intake down.
Remember this, also: These experiments were done only with initial fat deposits, newly formed in the blood vessel cells. There had been no chance for the deposit to become “set,” or for secondary changes to occur.
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